By V. Becker (auth.), Hans G. Beger MD, FACS, Markus Büchler MD (eds.)
Offers findings on acute pancreatitis within the fields of morphology, epidemiology, pathophysiology, enzymatic and in- strumental prognosis, scientific points and pharmacological and surgical procedure. Emphasis on scientific implications at the neighborhood unlock and vasoactice and poisonous components, the excessive expense of bacterial an infection within the necroses, and the factors of impairment of cardiocirculatory, pulmonary and re- nal organs.
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Extra info for Acute Pancreatitis: Research and Clinical Management
The following experiments were performed. 5 mM GDe to increase duct permeability. 5 mllh) in each case. 3 flg/kg body wt. 5 mM GDe and activated pancreatic juice. Group 2 comprised six cats in which histamine was administered intravenously (2 flg/kg body wt. 5 mM GDe and activated pancreatic juice. After the duct perfusions, the cannula was removed and the duct was ligated, the isoproterenol or histamine stopped, the abdomen closed, and the animal allowed to recover. Twenty-four hours later the animals were anesthetized again, Changes in Duct and Vascular Permeability 51 the pancreas was removed for pathological examination, and then the animals were killed.
This last finding also seems to be confirmed by the mortality rate (or percentage case mortality): in fact corresponding to the figure of 10% reported in the American series (probably influenced by the insertion of subjects with acute relapsing pancreatitis), we find in the English series a mortality rate that has remained practically unchanged in the last 10 years at around 20%. 28 G. Cavallini et aI. Sex Distribution When only the patients with a first attack of acute pancreatitis are considered, females appear more often than males.
Penny Roon and Ms. Allison. Ator for technical assistance. This paper was published in modified form in
Acute Pancreatitis: Research and Clinical Management by V. Becker (auth.), Hans G. Beger MD, FACS, Markus Büchler MD (eds.)